Femoroacetabular Impingement: Arthroscopic Management of the Proximal Femur



Femoroacetabular Impingement: Arthroscopic Management of the Proximal Femur


Christopher M. Larson

Rebecca M. Stone



Introduction

Femoroacetabular impingement (FAI) is a disorder characterized by abnormal conflict between the pelvis and proximal femur (1,2,3,4,5,6). Cam-type impingement is traditionally described as an offset pathomorphology of the femoral head–neck junction (1,2,3,4,5,6). Although the primary focus of cam-type FAI in the literature focuses on developmental decreased femoral head–neck offset, proximal femoral impingement can also be secondary to isolated femoral neck retroversion, coxa vara, slipped capital femoral epiphysis (SCFE), deformity secondary to femoral neck fractures, or trochanteric impingement which is classically seen in the setting of Legg–Calvé–Perthes disease (LCP) (1,2,3,4,5,6,7,8). The following chapter will focus on the indications and specific surgical techniques for arthroscopic management of the proximal femur in the setting of FAI. Anatomy and pathomechanics, epidemiology, typical patient presentation, appropriate imaging, and outcomes for arthroscopic treatment of cam-type FAI will also be discussed.


Anatomy and Pathomechanics

Proximal femoral impingement is typically the result of an aspheric femoral head and/or insufficient head–neck offset. Although this was originally termed a “pistol-grip” deformity and felt to be secondary to a mild or subclinical slipped capital epiphysis, further research has shown that this may be the result of increased lateral extension of the epiphysis (6). The pathogenesis of cam-type FAI is unclear. It has been suggested that excessive loading secondary to athletic activities during adolescence may result in adaptive physeal remodeling and bony apposition at the head–neck junction leading to this asphericity (9). This has been our observation in young adolescent athletes involved in high impact, cutting and pivoting activities. We have seen this most frequently in young male and female butterfly hockey goalies, who find themselves in repetitive high loading and extreme flexion, abduction, internal rotation positions at a young age. There also appears to be a genetic component as one study found that siblings of patients with cam-type FAI had a relative risk of 2.8 of having the same deformity compared to normal controls (10).

Although cam-type FAI is typically located anteriorly, it is important to recognize that there is much variability in the size and extent of these lesions. They can be primarily anterior, superior, posterior, inferior, or circumferential in some cases. The location has importance with respect to motion limitations and respective pain-generating activities, as well as with regard to choosing the most appropriate surgical approach when indicated. Cam-type FAI can also be seen in other situations. Residual deformity secondary to prior SCFE, LCP, or femoral neck fractures can lead to cam-type FAI (5,7,8) (Fig. 47.1). Excessive coxa vara and relative femoral neck retroversion can also contribute to proximal femoral impingement. In some situations, the greater trochanter can impinge against the pelvis and this is most frequently seen in the setting of LCP (5).

The abnormal femoral head–neck junction with its increased radius creates intra-articular damage as it is forced into the acetabulum during impingement range of motion (1,2,4,5,6,9). Isolated cam-type FAI generally produces shear forces, resulting in eventual disruption of the acetabular articular cartilage from the labrum (1,2,4,5,6,9). This labrochondral disruption may be partial thickness early on and can progress to large full-thickness chondral delaminations and eventual full-thickness exposed lesions over time (Fig. 47.2). It has been suggested that FAI may be a significant etiology leading to hip osteoarthritis (OA) (2,5). Many individuals with FAI, however, have a combination of cam- and pincer-type FAI and damage resulting from both mechanisms may be appreciated in these situations.


Epidemiology

The reported prevalence of radiographic FAI is variable depending on the specific patient population being studied
(10,11,12,13,14,15,16). One study evaluated radiographs of 817 patients of Asian descent with documented OA and reported only a 0.6% prevalence of FAI (14). Another study, however, looking at a cohort of 3,620 patients with OA from a Danish database reported a prevalence of cam-type FAI in 19.6%, and mixed-type FAI in 2.9% of males (15). More specifically, cam-type FAI has been reported to be most common in young athletic males (2,5,11,12,13). One study evaluated 200 asymptomatic Canadian volunteers with a mean age of 29 years, and found a 14% prevalence of cam-type FAI with 79% of those being male (11). Another study looked at 2,081 healthy young adults and found a 35% and 10% prevalence of cam-type FAI in males and females, respectively (13). It has been our observation that the prevalence of FAI and in particular cam-type FAI is higher in a young athletic population. In fact, one study evaluated radiographs of 34 athletes who presented with long-standing adductor-related groin pain and reported a radiographic prevalence of FAI in 94% of these athletes (16).






Figure 47.1. A: Radiograph of a 21-year-old male reveal cam-type FAI (arrow) secondary to a previous SCFE that was previously treated with internal fixation in situ. B: Radiograph of a 16-year-old female reveals an aspherical femoral head, acetabular dysplasia, and greater trochanteric overgrowth secondary to LCP.






Figure 47.2. A: Intraoperative image of the left hip reveals a partial-thickness labrochondral separation (arrow) secondary to cam-type FAI. B: Intraoperative image of the left hip reveals a full-thickness labrochondral disruption (arrows) secondary to cam-type FAI. F, femoral head; L, labrum.



May 22, 2016 | Posted by in PHYSICAL MEDICINE & REHABILITATION | Comments Off on Femoroacetabular Impingement: Arthroscopic Management of the Proximal Femur

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