(1)
Orthopaedics, Trauma and Plastic Surgery, University Hospital Leipzig, Leipzig, Germany
1.1 History
1.1.1 Localization and Nature of Complaint
Pain | Localization | Circumscribed/radiating |
Character | Numb/sharp/drilling/throbbing/burning/cramp-like | |
Situation | During certain movements, underload/at rest, elicited by sneezing, coughing, pressing, at night | |
Swelling | Location/extent | |
Deformity | Location/kind | Altered posture, deformity |
Movement disorder | Location/kind/direction | Limitation of movement/locking/stiffness, walking distance |
Sensory disorder | Location | Paresthesia/numbness/tingling/pins and needles |
Motor disorder | Location/degree (extent) | Muscular atrophy |
Uncertain gait/coordination disorders | ||
Location/degree (M5–M0a) | Weakness/paralysis/spasticity | |
Vertigo (cervical spine) | Rotating vertigo, lift vertigo | |
Motor function (cervical spine) | Swallowing disorders | |
Arm raise/lowering | ||
Elbow flexion and extension | ||
Making a fist/finger extension | ||
Motor function (thoracic/lumbar spine) | Hip flexion/extension, abduction/adduction | |
Knee flexion/extension | ||
Foot raise/lowering | ||
Bladder and rectal dysfunction |
1.1.2 Time Correlations
Beginning and course | Congenital/acquired (age/point in time) |
Acute/chronic | |
Slowly/stably progressive | |
Occurrence in flares with or without symptom-free intervals | |
During the day/at night |
1.1.3 Concomitant Circumstances
Accident | Yes/no | |
Location | Leisure/workplace | |
Mechanism of injury | Kind | Working overhead/lifting a load/heavy lifting/accident |
Triggering event | No triggering event/fall/bending/rising from a squatting position | |
Height/weight/load/persons involved | ||
Motor vehicle accident | Type of vehicles involved/direction/speed, angle of impact, lateral, head-on or rear-end collision, head impact/headrest/safety belts | |
Pre-existing conditions | Family medical history | |
Degenerative/bacteria/rheumatic inflammatory | No/if yes: local/systemic | |
Bacterial infection/viral infection | ||
Traumatic/tumor | ||
Malformations | ||
Common symptoms | Fever, weight loss (time period of weight loss), fatigue, nocturnal sweating | Yes/no |
1.1.4 Existing Treatment/Past Treatment
Drugs | Medication/dose/duration of intake | Localized/systemic |
Relief | Yes/no | |
Physical therapy | Application | Forms/duration/frequency |
Relief | Yes/no | |
Orthopedic aids | Walking stick/crutches | Yes/no |
Basques/bandage | ||
Brace/cast | ||
Operations | Time/site/type/success |
1.2 Cervical Spine
1.2.1 Systematic Examination
Local findings
Deviation from perpendicular line | Steep/overhang | Right/left |
Axis/position | Facial scoliosis | None; right/left convex |
Torticollis | None; right/left | |
Lordosis | Normal/flattened/kyphosed/prominence processus spinosus C7 | |
Shoulder position | Shoulder on equal level/elevated shoulder | Right/left |
Swelling/redness/heat | No; if present, then: | Localization/extent/scope |
Hematoma/abrasion/open wound/scab | No; if present, then: | Localization/extent/scope |
Scarring | No; if present then: | Localization/extent (soft/rough/displaced) |
Muscles | Paravertebral muscles | Highly developed/wasted/shortened |
Shoulder and neck muscles | Atrophy (significant?), myogelosis/muscle tone increase or decrease | |
Mobility | Chin–sternal notch distance: at anteflexion/at reclination | –cm/–cm |
Sideways tilting | −−/−−/−− degrees | |
Rotation | In neutral position (0 position) −−/−−/−−degrees | |
In anteflexion −−/−−/−− degrees | ||
In reclination −−/−−/−− degrees | ||
Tenderness | None; if present: | Suboccipital/spinous/interspinous/paravertebral (level; right/left) |
Tenderness upon striking | None; if present: | Level: spinous/interspinous |
Compression pain | Yes/no | |
Traction test | With pain relief/without pain relief |
Neurology
Horner’s triad | Absent/present | Right/left |
Deep tendon reflexes | Biceps (C5) Triceps (C7) Brachioradialis (C6) | Right/left Vigorous/decreased/absent/supernormal (hyperreflexic) |
Sensory examination | Dermatome (segment assignable/not exactly assignable) | Hypesthesia/paresthesia/dysesthesia |
Motor function examination | Shoulder abduction (C5/6) Elbow flexion (C5/6) Elbow extension (C7) Pronation (C6–Th1) Supination (C5/6) Wrist extension (C6/7) Wrist flexion (C6–Th1) | Right/left intact/impaired function (M0–M1–M2–M3–M4–M5) |
Circulation
Arteries | Radial artery | Fully/barely/not palpable (right/left) |
Vertebral artery: de Kleijn test | Negative/positive (right/left) | |
Carotid arteries: Adson test | Negative/positive (right/left) | |
Veins | Venous stasis | Present/absent (right/left) |
Capillary pulse | Fingertips | Visible/invisible |
Fig. 1.1
The clinician must look for any deviation from the imagined perpendicular line or asymmetry in the cervical spine. Clinical inspection also involves looking for possible muscle atrophy of the shoulder, neck, and paravertebral muscles. This figure illustrates atrophy of the right supraspinatus muscle
Fig. 1.2
(a, b) Testing the mobility of the cervical spine for anteversion (a) and retroversion. (b) (Normal values 35–45/0/35–45°). Alternatively, the values are recorded in cm between the chin and the sternal notch at maximal anteflexion (a) and maximal retroflexion (b)
Fig. 1.3
(a, b) Testing the sideways tilt of the cervical spine. (a, b) Normal values right/left 45/0/45°
Fig. 1.4
(a–c) Rotation of the cervical spine in neutral position (a), maximum retroflexion (b, concerns the lower cervical spine) anteflexion (c, concerns the upper cervical spine). Normal values for rotatory range of motion of the cervical spine: right/left, 60–80/0/60–80°
Fig. 1.5
Testing tenderness to pressure in the cervical spine (compression test)
Fig. 1.6
Traction test of the cervical spine (with or without pain relief)
Fig. 1.7
Horner’s triad left: ptosis, miosis, and enophthalmus. Horner’s triad can accompany a Pancoast tumor, lower plexus paralysis (Klumpke), and injuries of the thoracic and cervical spine
Fig. 1.8
Testing the biceps reflex. The arm is slightly flexed, and the examiner hits his own fingers placed on the distal biceps tendon with the reflex hammer
Fig. 1.9
Testing the brachioradialis reflex
Fig. 1.10
Testing the triceps reflex
Fig. 1.11
Segmental, sensory innervation of the upper limbs and the trunk (ventral and dorsal)
Fig. 1.12
De Kleijn test: patient lies prone on the examination table. Passive retroflexion, tilt, and rotation of the cervical spine to the affected side. After a few seconds, the patient complains of vertigo, caused by a compression of the vertebral artery of this side (the lower side)
Fig. 1.13
Adson test: The patient turns his head to the painful side while inhaling deeply. The examiner moves the arm of the same side backward and downward. The test is positive if the pain increases, if sensory disorder occurs, if the radial pulse weakens, or if stenosis sounds can be heard over the subclavian artery
1.2.2 Leading Symptoms of the Cervical Spine
The leading symptoms of the cervical spine are summarized in Table 1.1.
Table 1.1
Leading symptoms of the cervical spine
History | Pain | Local findings, functional tests | Sensory function disorder | Motor function disorder | Points to: |
---|---|---|---|---|---|
Incorrect load bearing, slight distortion, acute pain | Local neck pain | Painful limitation of the range of motion, localized tenderness to pressure | Normally none | Normally none | Acute cervical spine syndrome |
Chronic strain (computer work, secretary), chronic pain | Neck pain | Painful at the end of the movement, limited range of motion, paravertebral tenderness | Normally none | Normally none | Chronic cervical spine syndrome |
Chronic strain (computer work, secretary), headache, vertigo, chronic pain | Cervical and occipital radiating pain | At the end of the movement painfully limited mobility, nystagmus, suboccipital tenderness | Normally none | Normally none | Cervicocranial syndrome |
Chronic strain (computer work, secretary), chronic pain | Radiating pain in the shoulder, neck, and arm, not assignable to a specific segment | At the end of the movement painfully limited mobility, esp. the rotation at max. reclination, tenderness to pressure at the lower cervical spine, myogeloses | Normally none, dysesthesia not segmental | None | Cervicobrachial syndrome |
Nocturnal pain or post-strain pain | Radiating shoulder and neck pain, ulnar forearm, lateral edge of the hand, 4th and 5th fingers | Adson’s sign is positive (turning the head to the affected side) | Hypesthesia ulnar forearm, lateral edge of the hand, 4th and 5th fingers | Later failure of small hand muscles with atrophy and paresis | Scalenus syndrome DD: costoclavicular syndrome |
Pain, especially at night and after exercise | Radiating shoulder and neck pain, ulnar forearm, lateral edge of the hand, 4th and 5th fingers | Positive modified Adson test (turning the head to the healthy side) | Hypesthesia ulnar forearm, lateral edge of the hand, (4th and 5th fingers) | None | Cervical rib |
Acute, usually chronic pain, asthenia and sensory disturbances | Radiating pain in the shoulder, neck, and arm, to assign segmental, coughing, sneezing, and pressing pain | Positive cervical compression test, limited mobility, reflex deficit: biceps reflex (C5), brachioradialis reflex (C6), triceps reflex (C7) | Paresthesia possible, segmentally assignable: M. deltoideus (C5), the radial arm (C6), finger 2–4 (C7), little finger (C8) | Paresis possible: shoulder abduction (C5), flexion elbow (C6), extension elbow (C7), flexion hand (C7), adduction and abduction of the fingers (C8) | Radicular cervical compression syndrome (rare!) |
Unbalanced one-sided stress, slowly progressive weakness in the arms and/or legs, unsteady gait | Diffuse, partly radicular pain | Restricted mobility, increased reflexes | Uncharacteristic (also radicular or similar paraplegia!), paresthesia in hands and feet | Possibly voiding disorders, paresis (symmetrical, asymmetrical) tetra- or paraspasticity, flaccid paralysis of the hand (so-called myelopathic hand) | Chronic vertebral cervical myelopathy |
Intrauterine predicament, birth trauma | Mild pain | Head inclined to the healthy side, rotated to the diseased side | None | None | Muscular torticollis |
Childhood, combined with other malformations, deafness (rare) | Only in old age, radiating pain into the back of the head and arms | Short neck, low hairline, restricted mobility, elevated scapulae | None | Facial palsy (rare) | Klippel–Feil syndrome |
Juvenile rheumatoid arthritis, long-lasting rheumatoid arthritis, sensation of stiffness | No pain or resting and exercise pain, radiated to the mandible and occiput; radicular pain | Unsteadiness, rotation in anteflexion limited pain | Paresthesias in hands and feet | Weakness of the upper extremities, flaccid paralysis of the hands (myelopathic hand) | Atlanto-occipital instability |
Resistant to any therapy (suspected) cervicobrachial, Tietze, or impingement syndrome | Local pain in the anterior chest wall radiating into the arm | Tense shoulder and neck muscles, restricted mobility, Horner’s triad | Dysesthesia possible | None | Pancoast tumor |
1.2.3 Disorders of the Cervical Spine
Clinical presentations of disorders
Wry Neck (Torticollis)
Fixed misalignment of the head of a neonate or infant. The head is tilted to the diseased side and rotated to the healthy side.
Etiology: Due to intrauterine positions or birth trauma, a connective tissue shortening of the sternocleidomastoid muscle occurs. Genetic factors are being discussed.
Patient history: Movement impairment and malpositioning of the head.
Examination: Tilting of the head to the diseased side and rotation toward the healthy side. Occasionally swelling of the sternal muscle insertion. Hardening and shortening of the diseased muscle. Eventually facial scoliosis develops.
Diagnostics: Cervical X-ray in two levels to exclude a bony torticollis or a basilar invagination.
DD: M. Klippel-Feil, basilar invagination, ocular torticollis.
Acute and Chronic Cervical Spine Syndrome
This syndrome describes pain in the region of the cervical spine. If the pain radiates toward the arms, it is called cervicobrachial syndrome; if it radiates toward the back of the head, it is called cervicocranial syndrome. One differentiates between chronic and acute cervical syndrome.
In cervicobrachial syndrome, the segments C4 to C7 are affected. In cervicocranial syndrome, the segments C0 to C3 are affected. The vertebral artery and the sympathetic nerves can also be affected.
Etiology: Acute and chronic blocks in the small intervertebral joints as well as wear found in most degenerative diseases can be seen as the cause. Acute pain can set in after whiplash injury or after longer incorrect load bearing as well as exposure to cold draughts. Further causes are infections (rheumatoid arthritis) and tumors (rare). The etiology of cervicocranial syndrome is diverse. An interdisciplinary investigation including neurology, ENT, and internal medicine is always required.
Patient history: Acute (days, weeks) or chronic neck pain that originates from the cervical spine. If chronic, the pain often radiates into the arms (cervicobrachial syndrome). In this case, patients complain of dysesthesia. Pain that radiates into the back of the head (cervicocranial syndrome) is often accompanied by rotating vertigo and painful impairment of cervical movement.
Examination: Paravertebral tenderness on the affected segments, suboccipital and/or at the upper margins of the trapezoid muscle. In part increased muscle tone in the paravertebral muscles and myogelosis. Deviations from the perpendicular (Fig. 1.1). Movement impairments (rotation and sideways tilt) often unilateral (Figs. 1.2, 1.3, and 1.4). In the case of simple blocks, the traction test brings relief (Fig. 1.6). After whiplash injury, the cervical spinal can be tender when compressed (Fig. 1.5). Pain that radiates into the arm, as well as dysesthesia, can often not be pinpointed to certain segments. When the upper segments are involved, a gaze-evoked nystagmus can often be seen. The deep tendon reflexes are normal.
Diagnostics: X-ray of the cervical spine in two planes and in the semi-oblique projection for degenerative changes.
DD cervicocranial syndrome: Vertebral artery syndrome, de Kleijn (Fig. 1.12) positive—patient lies prone on the examination table. Passive retroflexion, tilt, and rotation of the cervical spine to the affected side. After a few seconds, patient complains of vertigo, caused by a compression of the vertebral artery of this side.
Neurological conditions
Radicular Compression Syndrome in the Cervical Spine
Cervical spinal root compression syndromes are characterized by radiating shoulder and neck pain that can be traced back to certain segments and are often accompanied by paresthesia. Radicular compression is less common in the cervical spine compared to the lumbar spine.
Etiology: Compression of a nerve root due to degenerative enlargement of the uncinate process or due to disc herniation or protrusion.
Patient history: Acute (rare) or chronic (more frequent) shoulder and neck pain. Pain that radiates up to the arms. Sometimes paresthesia. Pain upon coughing, sneezing, and pressing.
Examination: (Acute) Wry neck. Gaze-evoked nystagmus (upper segments). Suboccipital and paravertebral tenderness. Positive compression test (Fig. 1.5). Movement impairment of the cervical spine. Sensory loss and paresis. Absent reflexes or side difference.
Increased deep tendon reflexes, spastic muscle tone, and urinary incontinence (paraplegia symptoms) are highly suspicious of spinal disc herniation.
Somatosensory loss (. Fig. 1.11 ) that can be traced back to a certain dermatome is a sign of damage to the specific root. The region of the deltoid muscle is supplied by C5; the outer part of the upper limb and the radial part of the lower limb including the thumb are supplied by C6. Lateral and dorsal to C6 and the fingers II–IV are supplied by C7. The ulnar hand and the little finger are supplied by C8.
Motor loss of the C5 root is expressed in weakness in shoulder abduction and elbow flexion. Damage to C6 leads to an isolated paresis of flexion elbow. Weakness in the flexion of the hand and extension of the elbow occurs when C7 is damaged. A weakness in the abduction; adduction is connected to C8 damage.
Absent or depressed reflexes can be a neurological sign of root damage.
The biceps reflex (Fig. 1.8) communicates with the C5 root, the brachioradialis reflex with (Fig. 1.9) C6, and the triceps reflex with (Fig. 1.10) C7 und C8.
Diagnostics: X-ray of the cervical spine in two planes and semi-oblique to rule out bony alterations. CT and MRI to prove disc herniation or protrusion. Neurological consultation including possible EMG.
DD: Neuralgic shoulder amyotrophy, carpal tunnel syndrome, compression of the ulnar nerve.
Chronic Cervical Myelopathy Deriving from the Vertebra
This disease describes a mostly unilateral, slowly progressive weakness of the arms and/or the legs caused by a stenosis of the spinal canal at one or more levels. The levels C4–C7 are frequently affected.
Etiology: Mostly due to osteophytes (cervical spondylosis). Cervical myelopathy can also occur in rheumatic disease involving atlanto-occipital instability.
History: Often chronic neck pain. Weakness and a sensation of heaviness in the arms and/or the legs, often unilateral. Gait instability, diffuse, but also radicular pain in the arms and legs. Bladder or urinary problems are rare.
Examination: Reduced mobility of the cervical spine (Figs. 1.2, 1.3, and 1.4). Sensory loss is not characteristic (mostly absent, but can also present similar to losses seen in radicular or spinal trauma). Motor loss in the form of paresis (symmetrical or unilateral) or spastic tetra- or paraplegia. Deep tendon reflexes (Figs. 1.41 and 1.42) are increased; the Babinski sign is positive (Fig. 1.43), possibly also the Gordon and Oppenheimer signs (Figs. 1.44 and 1.45). The “myelopathic hand” could also possibly be seen: thenar, hypothenar, and interosseous muscle atrophy.
Diagnostics: X-ray of the cervical spine in two planes and semi-oblique. CT and MRI to visualize bony stenosis and to exclude disc herniation or protrusion. Neurological consultation (EMG).
Lesions of the Cervicobrachial Plexus, C1–T1 (T2)
Divided into lesions of the cervical plexus and lesions of the brachial plexus. The brachial plexus is further divided in the upper, the whole, and the lower plexus lesions.
Diagnostics: As with all plexus lesions, neurological examination and EMG is required. This serves to verify the damage and the extent of the lesions. Furthermore, myelography (bloody cerebrospinal fluid or an “empty root pouch” as a sign of root lesion).
Especially in the case of traumatic damage, the clinical presentation can initially be inhomogeneous. This calls for a repeat of the clinical examination.
Sweat secretion upon pilocarpine or a histamine test (local flush reaction). For this one must consider that the sympathetic nerves originate below T3. If the vegetative innervation of the upper extremity is intact (sweat secretion, localized flushing), root damage is almost certain. Plexus lesions can, however, also be combined with root damage.
Lesions of the cervical plexus (C1–C4)
Relatively rare, because the plexus is anatomically well secured. Patients rarely survive trauma in this region. Tumors, inflammation, and radiation damage rarely present in this region.
Brachial plexus lesions ((C4) C5–Th1 (Th2))
Lesions of the root or the plexus.
Etiology: Direct trauma (e.g., motorcycle accidents); iatrogenic (perioperatively due to patient positioning in anesthetics, brachial plexus block), birth trauma, inflammation, radiation damage, compression syndrome, neuralgic shoulder amyotrophy.
Upper brachial plexus lesion (Duchenne–Erb)
Most common monoplegia. Affects C5/C6.
Etiology: Mostly caused by trauma, including birth trauma.
History: Patients are unable to use the shoulder to lift the arm away from the trunk. The arm hangs flaccid and the hand is rotated inward.
Examination : Shoulder, active abduction and outer rotation M0; elbow, active flexion and supination M0. Hypesthesia (Fig. 1.11) over the deltoid muscle and the radial side of the lower arm.
Whole lesion of the upper brachial plexus
Paralysis similar to Duchenne–Erb. Yet includes, beyond C5–C6, also C7. (The singular lesion of C7 is also referred to as mid-arm plexus lesion. C7 lesions can also occur in combination with lower brachial plexus lesions.)
History: Arm lift impossible. Arm hangs flaccid and rotated inwardly. Weakness in the arm and hand.
Examination: Shoulder, active abduction and outer rotation M0; elbow, active extension/flexion as well as pronation reduced. Hypesthesia (Fig. 1.11) over the deltoid muscle and the radial side of the lower arm and the hand.
Lower brachial plexus lesion (type Déjerine–Klumpke)
Relatively rare. Paralysis of C8 and T1. Includes paralysis of the lumbrical muscles, the long finger flexors, and in part the long hand flexors.
History: Drastic functional impairment of the hand.
Examination: Paresis of the small hand muscles (e.g., lumbrical muscles) and the long finger flexors. Ulnar claw (Fig. 2.46). Sensory loss on the ulnar side of the lower arm and hand is always present under cervical impingement syndrome (Fig. 1.11). Often accompanied by Horner’s syndrome (Fig. 1.7).
Impingement Syndromes
Chronic damage to the lower parts of the brachial plexus. Can be accompanied by stenosis of the local blood vessels.
Etiology: Compression at anatomically narrow areas.
Examination: Provocation test (Adson test); not very specific, also positive in healthy persons. X-ray of the cervical spine in two planes and of the shoulder in two planes. Neurological consultation (EMG, nerve conduction velocity (NCV)). Doppler sonography, angiography (DSA).
DD: Pancoast tumor (pain-free); neuralgic shoulder amyotrophy; “backpack palsy” (Fig. 1.24)(due to carrying a heavy load on the shoulders); less common is thrombosis in the axillar vein, occlusion of the brachial artery.
Cervical rib
Additional rib at C7
Etiology: Congenital.
History: Shoulder and neck pain especially at night and upon exertion. Numbness and pain can be found in the ulnar lower arm, the lateral hand, and the fourth and fifth digit.
Examination: Pain can be provoked by turning the head to the healthy side. This causes loss or weakening of the radial artery pulse and hypesthesia in the fourth and fifth digit (Fig. 1.11).
Scalenus syndrome
Impingement of the subclavian artery and the brachial plexus in the course of their exit through the frontal scalene hiatus. Can lead to motor function loss in the small hand muscles, including atrophy and paresis.
Etiology: Enlarging of the anterior scalene muscle at its insertion sight.
History: Brachialgia, paresthesia, sensory loss. In the course of disease, weakness of the hand.
Examination: Positive Adson test (Fig. 1.13). This test involves turning and lifting the head toward the affected side while the patient simultaneously inhales deeply and the ipsilateral arm is pulled in a caudal direction. Sensory loss in the fourth and fifth digit and the lateral hand. Finger abduction and adduction are limited.
Costoclavicular syndrome
Compression of the brachial plexus, the subclavian artery, and the subclavian vein between the clavicle and the first rib.
Etiology: Partly congenital, improper healing of clavicle fractures (fibrocartilage callus), tumors in the clavicle.
History: Brachialgia, paresthesia, sensory loss.
Examination: Positive Adson test (Fig. 1.13). This test involves turning and lifting the head toward the affected side while the patient simultaneously inhales deeply and the ipsilateral arm is abducted up to the horizontal plane and pulled backward. Sensory loss in the fourth and fifth finger and the lateral hand.
Hyperabduction syndrome
Rare. Compression of the blood vessels and nerves between the coracoid process and the minor pectoralis minor muscle.
Etiology: Regular hyperabduction of the arm, often in a context of work or sport (builders, painters, javelin).
History: Brachialgia, especially during the abovementioned actions, also during sleep with a cranially extended arm.
Examination: Pain provocation by passive retroversion of the arm in maximum elevation. Weakening of the radial pulse. Motor or sensory loss rare.
Congenital malformation
Klippel–Feil Syndrome
Congenital fusion of two or more cervical vertebrae. Often associated with other dysplasia, especially in the upper limb (syndactyly, hypoplasia, Sprengel anomaly) but also of the internal organs (kidney, heart).
Etiology: Unknown.
History: Localized or radiating pain the arms and the back of the head. This can often occur in old age due to premature wear of the vertebrae. Often reduced range of motion in the cervical spine.
Examination: A short neck and low hairline is often seen. Scoliosis is common as well as in combination with the Sprengel anomaly (elevated shoulder blade). Sometimes the dysplasia of the cervical spine causes a bony wry neck. Facial nerve palsy or deafness is rare.
Diagnostics: X-ray of the cervical spine in two planes and semi-oblique. Eventually functional imaging. Neurological consultation (EMG, NCV) and internal medicine consultation (dysplasia of the internal organs).Stay updated, free articles. Join our Telegram channel
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