Orthoses for overuse disorders of the upper limb

Chapter 21 Orthoses for overuse disorders of the upper limb




Our hands are the masters of many functions, ranging from delicate touches to forceful grips. We use our hands to lift, pull, push, pinch, and grasp. The hand is an instrument needed for building, communicating, feeding, and expressing emotion. Whereas a closed fist denotes anger, an open palm offers comfort and empathy. The blind use their hands to read, and the deaf to talk. Our hands are unique tools required to perform all aspects of our activities of daily living. The many daily tasks in our lives can impart a demanding burden upon the hand and upper extremity. Repetitive motion, improper posture, and poor mechanics can lead to cumulative trauma disorders characterized by inflammation, pain, limited movement, nerve compression, and ultimately impaired function.


This chapter focuses on the conservative orthotic management of overuse disorders of the upper extremity. Many of these conditions will also require other conservative treatments, including, but not limited to, nonsteroidal antiinflammatory drug (NSAIDs), steroid injections, ergonomic modifications of work activities and the workplace, exercise, or modalities such as paraffin, hot pads, ice packs, iontophoresis, or ultrasound. Although a global approach to conservative treatment of these conditions is necessary and orthoses will be used in conjunction with other treatment options, this chapter discusses only the appropriate splints to be used with these overuse conditions.


In order to determine the type of splint necessary for any condition, it is important to understand the general goals of splinting. The ultimate purpose of orthosis in the upper extremity is to relieve pain and improve functional range of motion. Some of the general goals of splinting include (a) providing rest for a specific joint, (b) relieving tension from healing structures, (c) protecting and properly positioning edematous structures, (d) maintaining proper joint alignment and tissue length to prevent soft-tissue contracture, and (e) providing maximum functional use of the extremity while it is splinted. It is necessary for the physician and therapist to fully understand the anatomy of the involved structures and the anatomical implications of splinting to avoid pressure areas and compression of surrounding nerves and to prevent further inflammation of surrounding structures. If all of these factors are taken into consideration, splinting can be an effective option for conservative management of these nontraumatic conditions.



Lateral epicondylitis



Anatomy and symptoms


Lateral epicondylitis, or tennis elbow, is a very common condition of the lateral elbow. Patients generally present to the clinic with pain and localized tenderness of the extensor origin and lateral epicondyle of the humerus. Onset may be acute or insidious. Generally, the patient complains of increased pain with wrist extension or with gripping. In severe cases, the patient may complain of pain with lifting very light objects. Activities requiring full elbow extension and forearm pronation aggravate the discomfort. The pain of lateral epicondylitis often is accentuated by extreme wrist flexion from passive stretch of the extensor carpi radialis brevis (ECRB) muscle or by active contraction of the wrist extensors. The pain may not be isolated to the lateral epicondyle but actually may radiate proximally or distally.9,11,26,27 Lateral epicondylitis is believed to be a tendinosis of the origin of the ECRB. Although the specific etiology is unknown,2 it is postulated that microtears at the origin of the common extensor muscle mass, involving the extensor digitorum communis, extensor carpi radialis longus, supinator, and ECRB, are the likely precipitating factor for this condition. Many investigators believe that these small tears are caused by overuse and repetitive injury and strain of the common extensor origin at the lateral epicondyle. The instigating injury is the result of forced flexion of the wrist and fingers during extensor muscle contraction. Such mechanisms are seen with racquet sports where backhand volleys are required. As these microtears try to heal, continued use of the upper extremity causes continued strain and reinjury to the muscle origins.10,26,27 This can lead to degeneration and possible avascularity at the muscle origin, which in turn leads to chronic inflammation causing the injured areas to remain weak and painful.



Splinting lateral epicondylitis


The primary goal of splinting lateral epicondylitis is to decrease the pain and inflammation at the origin of the ECRB. Several splints have been described in the literature.2 One of the most common splints used is the wrist cock-up splint (Fig. 21-1). This rigid orthosis maintains the wrist in extension to offload the extensors of the forearm and promote healing at the muscle origin.10 The appropriate posture of the wrist in the splint has been described in various positions, ranging from 0 to 45 degrees of extension.4,10 Janson et al.10 compared three types of wrist orthosis—dorsal wrist splint, volar wrist splint, and semicircumferential wrist design—varying the amount of wrist extension and then evaluating muscle limitations by surface electromyography (EMG). The semicircumferential splint showed the greatest change in EMG electrical activity of the extensor muscles: a 6% decrease. The authors concluded that this reduction of electrical activity was comparable to the reduction of electrical activity of the wrist extensors seen while a proximal forearm counterforce brace was worn. Based on these findings, combined with the limited functional use while a rigid wrist orthosis was worn, the authors did not recommend wrist splinting. Clinically, we have found that compliance with a rigid wrist splint is poor because of the device’s functional limitations.



The forearm support band or counterforce brace (Fig. 21-2) is a common splint prescribed for lateral epicondylitis. The counterforce brace decreases the force of the muscle contraction by inhibiting muscle expansion and reducing tension at the musculotendinous unit proximal to the band.17 The counterforce brace essentially changes the functional origin of the extensor mass to a site distal to the radial head. This may assist in resting the origin of the common extensor tendon to permit healing.9,17



Several designs of forearm bands are on the market. Both standard and Aircast® counterforce braces showed reduced EMG activity of the extensor muscles, with the greatest reduction of EMG activity seen with the Aircast® band (Fig. 21-3).17 Further studies are needed to determine recommended wearing time and amount of pressure with application, but it is generally agreed that the counterforce brace should be worn for painful activities during the day and removed for any periods of inactivity. The brace typically is applied approximately 2 cm distal to the lateral epicondyle. It should be fit with a comfortable amount of pressure while the muscles are relaxed so that maximum contraction of the wrist extensors is prohibited. Potential complications with use of the counterforce brace include compression of the radial nerve or the anterior interosseous nerve, edema, and venous congestion. Clinically, we have found more positive compliance with the counterforce brace because it leaves the hand completely free for functional use.



In acute cases of painful lateral epicondylitis, an initial trial of complete immobilization may be necessary. An example is a long arm splint with the elbow in 90 degrees of flexion, the forearm neutral, and the wrist positioned in comfortable extension.27 The other splinting methods previously described can be initiated once the acute pain has been relieved to a functional level.



Cubital tunnel syndrome



Anatomy and symptoms


Ulnar nerve compression at the medial elbow is known as cubital tunnel syndrome. The cubital tunnel is a fibroosseous tunnel at the elbow situated between the humerus and ulnar heads of the flexor carpi ulnaris. The cubital tunnel is bordered laterally by the ulnohumeral collateral ligament and anteriorly by the medial epicondyle. The roof of the cubital tunnel, known as Osborne’s, is a fibrous band confluent with the two heads of the flexor carpi ulnaris starting just proximal to the medial epicondyle.22


The most common symptoms of cubital tunnel syndrome are paresthesias presenting as numbness and tingling of the ring and small fingers, especially notable at night. Patients may complain of tenderness or a sharp pain at the medial epicondyle or directly over the ulnar nerve. They may describe a vague, sometimes sharp, radiating pain down the ulnar forearm and into the hand. Symptoms may increase with provocative positions of elbow flexion or repetitive flexion/extension. Late stages or severe cubital tunnel syndrome may present with motor weakness and/or muscle atrophy of ulnar innervated muscles, causing a decrease in pinch and grip strength. Ulnar clawing and intrinsic muscle wasting may be present in late stages of cubital tunnel.


Prolonged elbow flexion or repetitive elbow flexion and extension can be a major causative factor to cubital tunnel symptoms. Apfelberg and Larson1 described the condylar groove, where the ulnar nerve passes, as rounded and spacious during extension and flattened and triangular with elbow flexion, causing increased pressure on the ulnar nerve. Rayan et al.21 showed that elbow flexion produces a significant rise in pressure at the condylar groove, and pressure increases further at the cubital tunnel when elbow flexion is combined with shoulder elevation.22,24 Many patients experience an increase in symptoms at night because their elbows are flexed during sleep. Other potential causes of cubital tunnel syndrome include elbow injury and inflammation, and direct trauma or pressure to the ulnar nerve. Ulnar nerve subluxation or traction also may cause symptoms.



Splinting cubital tunnel syndrome


Patients with mild symptoms and no motor changes generally respond well to conservative splinting. The primary goal of splinting for this condition is to decrease pain and paresthesias. Immobilization with the elbow in extension decreases the stretch on the ulnar nerve and prevents repetitive motion that can promote an inflammatory response within the cubital tunnel. An elbow pad can be used during the day to protect the ulnar nerve from trauma or direct pressure.24


Night splinting is essential to prevent sleeping with the elbow flexed beyond 90 degrees. Splints for wear at night can be a custom-fit device or an off-the-shelf type pillow splint. Full elbow extension allows for the least amount of pressure on the ulnar nerve; however, most patients will not be able to tolerate immobilization in full extension. Therefore, a custom splint should be fit with the elbow in comfortable extension, anywhere between 30 and 60 degrees of flexion.3 The splint is generally fit anteriorly to avoid direct pressure on the nerve at the posterior medial elbow (Fig. 21-3). In our clinic, we have found this type of splint to be effective, yet the rigidity of the splint makes compliance questionable. For the elbow we tend to use a soft splint known as the Posey Soft SplintTM (Fig. 21-4) The Posey soft splint is a filled with polystyrene beads. The splint is easily applied with hook-and-loop closures and prevents elbow flexion. This splint is bulkier than the custom splint, but it is not rigid, is lighter and more comfortable, and improves patient compliance.




Carpal tunnel syndrome



Anatomy and symptoms


Carpal tunnel syndrome is the most common compression neuropathy of the upper extremity. The carpal tunnel is a small narrow space in the volar wrist that houses the median nerve, flexor pollicis longus, four flexor digitorum profundus tendons, and four flexor digitorum superficialis tendons as they course through the forearm into the hand. The tunnel is formed by the concave arch of the carpal bones and the transverse carpal ligament, which extends from the scaphoid tuberosity and trapezium radially, and attaches to the pisiform and the hook of the hamate on the ulnar side of the hand. The carpal tunnel has a defined space with little room for expansion. An increase in the volume of the contents of the tunnel will have a detrimental compressive effect on the tendons and median nerve, resulting in symptoms of carpal tunnel syndrome. Numerous causes of median neuropathy include inflammatory synovitis, trauma, repetitive flexion/extension, and autoimmune and endocrine disorders (Table 21-1).


Table 21-1 Factors involved in the pathogenesis of carpal tunnel syndrome





Anatomy

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Jul 12, 2016 | Posted by in ORTHOPEDIC | Comments Off on Orthoses for overuse disorders of the upper limb

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