Disorders of the wrist


Disorders of the wrist


Lesions at the wrist often have a traumatic cause – either a single injury or overuse – and are localized. Usually the patient can locate the lesion precisely because wrist and hand disorders seldom cause referred pain. If the symptoms appear spontaneously and are diffuse over the entire wrist, arthritis, usually of the rheumatoid type, is very likely. Further data obtained from the history include pain at rest, pins and needles, twinges and functional loss. The interpretation of the functional examination is classic and encompasses inert structures (capsular, non-capsular and ligamentous lesions) and contractile structures.

Disorders of the inert structures

Limited range: capsular pattern

The capsular pattern is an equal degree of limitation of flexion and extension (Fig. 23.1). In advanced arthritis, fixation of the wrist in the neutral position takes place.

Traumatic arthritis

The discovery of a capsular pattern following a trauma should always induce further examination. Uncomplicated traumatic arthritis is rare and the finding of a post-traumatic capsular pattern is highly suggestive of a fracture. The radiographic picture is unreliable because it may take up to several weeks for radiological evidence of a fracture to be conclusive.1 This delay may be responsible for serious complications because failure to diagnose and immobilize carpal fractures often leads to non-union and avascular necrosis.2,3 However, the clinical picture is so evident that the diagnosis should not be missed: any case of traumatic arthritis of the wrist lasting for longer than 2 days should be considered as a fracture.

History and examination

The history is that of an injury, varying from a simple thrust to a fall onto the outstretched and dorsiflexed hand. There is instant and severe pain that prevents hand movement. Initially the pain is localized but spreads over the entire wrist later on.

Inspection shows swelling of the wrist. On examination, a capsular pattern is found: passive flexion and extension are very painful and limited. The end-feel is that of a vibrant twang caused by a protective muscle spasm. Passive deviation of the wrist towards the painful side also increases the pain – a compression phenomenon that provides further evidence of a bony lesion. Careful palpation of the bones reveals localized tenderness. Most often the scaphoid bone is involved.4 In this case, tenderness will be elicited in the anatomical snuffbox.5 This is often described as a classic sign of scaphoid fracture and has a reported sensitivity of 90% but a low specificity of 40%.6 The scaphoid compression test also has a high sensitivity but a better specificity for scaphoid fractures. This test is performed by grasping the thumb and applying an axially directed compressive force along the line of the thumb metacarpal; wrist pain indicates a positive result.7

The scaphoid bone spans the joint between the proximal and distal rows of carpal bones and is most vulnerable: scaphoid fracture comprises between 51 and 78% of all carpal fractures.8–11 Young males and persons between 10 and 19 years of age are at highest risk for scaphoid fracture.1214

Scaphoid fractures occur most easily during loading of a hyperextended wrist, possibly in combination with ulnar deviation. This movement tightens the palmar ligaments (the radiocapitate and radioscaphoid ligaments), so stabilizing the scaphoid against the radius. As the proximal pole is strongly stabilized, further loading and extension of the wrist causes bending forces on the distal pole, which may result in fracture.15,16

Fractures may occur at any level in the scaphoid but the more proximal the fracture, the greater the chance of avascular necrosis of the proximal fragment.17,18 The main reason for this is the special nature of the scaphoid’s blood supply.19 The blood supply to the scaphoid can be divided into extraosseous and intraosseous sources. The extraosseous blood supply is primarily derived from a branch of the radial artery, the artery to the distal ridge of the scaphoid.20 The branches of this vessel enter the scaphoid through a foramen at the dorsal ridge at the level of the wrist (Fig. 23.2). These vessels then run proximally and palmarly within the medullary chamber, forming the intraosseous supply to the proximal pole.21 Since vascularity of the proximal pole is limited and dependent on intraosseous flow, acute proximal pole fractures have a potentially prolonged healing period, averaging 3–6 months, and there is higher incidence of non-union.

Because of the danger of avascular necrosis developing, it is important to recognize a scaphoid fracture as quickly as possible. Acute scaphoid fractures are often missed on initial plain radiographs. Therefore an initial negative radiograph does not alter the clinical diagnosis. When clinical suspicion of a scaphoid fracture is high and plain films are negative, the traditional recommendation is for these patients to be immobilized in a thumb spica splint or cast, with repeat radiographs after about 2 weeks.22

Alternative imaging modalities for diagnosis include bone scans and magnetic resonance imaging (MRI). A bone scan reportedly shows focally increased uptake within 72 hours and is very sensitive for detecting a fracture, but may not be extremely specific.23–25 MRI has been reported to have 95–100% sensitivity and almost 100% specificity for scaphoid fractures.2628 Additional advantages of MRI include its potential to assess vascularity of the proximal scaphoid pole when a fracture is present.29


Treatment consists of immediate immobilization of the wrist, which is protective and can decrease the incidence of non-union and avascular necrosis.30 The best position remains unclear. Several dissection studies have come to different conclusions.3134 Immobilization seems to be best performed with a thumb–spica cast, holding the thumb in the palmar abducted position and the wrist in the mid-position but with an element of deviation towards the site of the fracture, which, for the scaphoid, would be radial deviation. This helps to press the fractured surfaces together and stimulates the healing process.15 The time needed for a scaphoid fracture to heal is variable, from a minimum of 6 weeks to as long as 9 months,35 but 90% of fresh fractures heal with adequate treatment.30

Fractures of the other carpal bones can occur. Frequently, multiple radiographic views and/or follow-up studies with tomograms or bone scans may be necessary for a definitive diagnosis.36 Displaced fractures may require open reduction and internal fixation.37,38

Rheumatoid arthritis

The wrist can become affected by any type of rheumatic disorder, such as systemic lupus erythematosus, progressive systemic sclerosis, psoriatic arthritis, gout and pseudogout, ankylosing spondylitis and sarcoidosis, but rheumatoid arthritis is one of the commonest, frequently affecting both wrists39 and often following involvement of the fingers. In the acute stage a capsular pattern is found on examination, together with swelling, warmth and synovial thickening. At this stage MRI can be a useful technique for reaching a diagnosis.40 Recently joint ultrasound has also proved to be an important tool to evaluate synovitis of the wrist.41 In addition to pain, the more chronic cases present with gross limitation of movement. At this stage, there is no longer any warmth and even the swelling has diminished; diagnosis is easily made on conventional radiographs.

In the acute stage, the patient can be treated locally by immobilization of the wrist for some weeks. In the subacute and chronic stages, triamcinolone is a much better approach. One infiltration of 20 mg of triamcinolone acetonide is given at all the different areas of synovial thickening and/or tenderness, which are first precisely identified by palpation. Although treatment is very painful, the results are spectacular and symptoms at the wrist tend not to recur.

Limited range: non-capsular pattern

Carpal subluxation

Subluxation of a carpal bone, most often the capitate, occurs quite frequently. The diagnosis is purely clinical and, once the condition is recognized, it can be dealt with successfully by conservative management. Recurrence is not at all uncommon and may ultimately cause problems.

History and examination

The patient complains of pain on certain movements at the dorsal aspect of the wrist, especially when weight is put on an extended wrist. The symptoms may come on spontaneously or as the result of a minor injury in flexion.

On examination, signs of internal derangement are found (Fig. 23.3): one movement, most often extension, is limited with certainly in acute cases an end-feel of muscle spasm. Other movements are full-range, although often painful the result of overstretching the irritated ligaments (see p. 34). The bony subluxation may be visible and palpable if the wrist is held in flexion. There is local tenderness in the ligaments.

The radiograph is negative: the edge of the subluxated bone appears merged with the others and cannot be visualized separately on the radiograph, nor can its position be measured against the superimposed margins of the other bones (Cyriax:43 p. 184).

The clinical examination supplies the diagnosis: the joint is blocked in one direction only, (which is typical of internal derangement) and there is a visible displacement on inspection. Instant reduction during manipulation confirms the tentative diagnosis.

Palmar subluxation, mostly of the lunate bone, is very exceptional. It gives rise to (slight) limitation of flexion and may compress a palmar branch of the median nerve, causing paraesthesia in the corresponding territory (see online chapter Nerve lesions and entrapment neuropathies of the upper limb).


Treatment consists of manipulative reduction, which restores extension. Because of the accompanying sprain of the dorsal ligaments, flexion often remains painful. A few sessions of deep transverse friction will resolve this problem. If the condition recurs, manipulation is repeated. Frequent recurrences indicate instability and should be treated with infiltration of a sclerosant solution into the ligaments attached to the subluxated bone.

Basic technique: manipulation for carpal subluxationimage

The patient sits on a couch, the arm held in abduction against the back of the couch. An assistant grasps the patient’s upper arm just proximally to the elbow and holds it firmly against the couch. One foot is placed forwards for the manipulator’s benefit: the latter will place a foot against it.

The manipulator stands level with the patient’s hip, between the couch and the patient’s arm, and places the ipsilateral foot against the assistant’s foot; later, this will enable the manipulator to lean sideways and use the body weight for the pull required to obtain traction on the wrist joint. With the ipsilateral hand, the patient’s forearm is grasped just proximally to the radiocarpal joint, with the thumb on the dorsal aspect of the forearm. With the contralateral hand, the patient’s wrist is held with the thumb dorsally on the subluxated bone.

The aim of the manipulation is to perform a gliding movement between the rows of carpal bones during traction. The technique is executed as follows. The manipulator leans sideways but makes sure that the pull is only carried out with the distal hand, so as to obtain traction on the wrist. After the slack has been taken up, both hands are moved vertically up and down in opposite directions (Fig. 23.4). The little finger of the contralateral hand is kept in the palm of the patient’s hand to control movement; the manœuvre should result in a pure gliding movement and flexion–extension should be avoided.

This gliding movement is performed several times in one manœuvre. The movements of the wrist are then re-examined and the manipulation repeated until a full range of extension has been restored or until no further benefit results. If there is no immediate result, the attempt should not be abandoned too quickly. It may sometimes help to slightly change the position of the patient’s hand during the manœuvre and to build in a bit more flexion, extension or radial or ulnar deviation.

When this technique does not lead to full recovery, the next manœuvre is tried.

Second technique: manipulation for carpal subluxationimage

This technique is never used first but is introduced only when the bone is partially reduced. The aim is to perform a squeezing of the wrist during traction.

The patient, assistant and manipulator maintain the same positions as in the previous technique. The manipulator’s distal hand is now moved slightly downwards, so that it grasps the patient’s hand at the level of the bases of the metacarpal bones. Using the other hand, the patient’s wrist is approached from above and encircled with thumb and index finger. The metacarpophalangeal joint of the index finger is placed on the subluxated bone. The manipulator’s forearm is held vertically (Fig. 23.5). The manipulator leans sideways, applying traction with the distal hand and, having taken up the slack, executes a short, sharp squeezing manœuvre.

Although the manipulative thrust is a pure squeezing, there is an involuntary downwards movement of the hand, which helps reduction. This technique can be repeated several times.

Alternative technique: manipulation for carpal subluxation

The patient stands next to the couch, with the forearm, stabilized by the other hand, placed on the couch, the distal part level with the edge. The manipulator stands in front of the patient and grasps the hand in such a way that one thumb, reinforced with the other, is placed dorsally on the subluxated bone, while one index finger, reinforced by the other, is placed on the palmar aspect of it. The little fingers are placed in the palm of the patient’s hand, in order to prevent flexion–extension movements during the manipulation (Fig. 23.6).

Traction is applied with both hands and, after having taken up the slack, an anteroposterior glide is performed with emphasis on downward movement. Several movements are performed and the patient is re-examined after each manœuvre.

Other joint problems

The four following disorders, all visible on radiograph, give rise to limited extension only.

Aseptic necrosis of the lunate bone (Kienböck’s disease)

Avascular necrosis of the lunate was first described by Peste in 1843.44 The condition was then forgotten until 1910, when Kienböck recorded lunatomalacia, which he assumed had a vascular traumatic aetiology.45 Despite recognition of this disease entity for the past 100 years, its cause is still debated. Some investigators relate it to a stress fracture that leads to devascularization of the major segment of the lunate. Other authors consider the avascular necrosis to be the result of interruption of the vascularization of the lunate bone following excessive shear forces caused by acute trauma or repeated microtrauma.46 This would happen more easily in those lunate bones that are supplied by a single volar or dorsal blood vessel.47 Others suggest that a vascular non-traumatic process, with a minor infarction pattern in the proximal subchondral area, may be nearer to the truth.48

Avascular necrosis usually affects the dominant hands of males between 20 and 40 years of age.49,50 The symptoms come on spontaneously or as the result of minor injury, and usually interfere very significantly with work-related activities. The initial symptoms are pain and stiffness, which may vary from moderate to severe and incapacitating. The patient may also mention reduced strength in the hand.51

Clinical examination in the initial stage shows a capsular pattern with muscle spasm, due to the accompanying synovitis. Later, painful limitation of passive dorsiflexion becomes obvious, together with pain at the end of range of the other movements – a clear non-capsular pattern.

The classification of Kienböck’s disease is based on its roentgenological appearance.52,49,53:

Treatment methods extend from immobilization to revascularization surgery on the affected bone. However, there is still no gold standard for the treatment of Kienböck’s disease.56 A recent systematic review showed that, to date, there are insufficient data to determine whether the outcomes of any intervention are superior to placebo or the natural history of the disease.57

Avascular necrosis in other carpal bones has been described but is rare.58,59

Full range

Although passive movements of the wrist show a full range of movement, they can be painful at the end of range, showing that a structure is either stretched (ligament) or pinched (impaction syndromes).

Ligamentous lesions

Ligamentous injuries range from minor sprains with no instability (which are discussed here) to complete rupture with gross instability (see p. 341).61

Sprain of the ulnar collateral ligament

This disorder is the result of an injury causing a fracture, either a Colles’ fracture or fracture of the styloid process of the ulna. The fracture has healed but pain remains at the ulnar aspect of the wrist. On inspection, the deformity of the radius is visible and passive radial deviation causes pain at the ulnar aspect of the wrist. The symptoms can be quickly relieved by an infiltration of triamcinolone.

Sprain of the radial collateral ligament

This rare disorder results in pain felt at the radial aspect of the wrist during passive ulnar deviation. A more common cause of these symptoms is a tendinous lesion in the extensor pollicis brevis and abductor pollicis longus tendons (see p. 354). Differential diagnosis is easy because, in tendinitis, resisted movements are also positive.

Radial collateral sprain responds well to treatment with either one infiltration of triamcinolone or several sessions of deep transverse friction.

Sprain of the dorsal ligaments

The most common minor lesions are usually the result of overuse but may also occur when long-standing carpal subluxation causes irritation of the overstretched ligaments.

Pain is felt at the dorsal aspect of the wrist on full passive flexion. Other tests are negative. Careful palpation reveals the exact site of the lesion. Most of the lesions are found in the lunate–capitate ligament but occasionally also in the radiolunate, capitate–third metacarpal or ulnar–triquetral ligament.

Treatment consists of several sessions of deep transverse friction, which should lead to complete cure, independently of how long symptoms have been present. In the rare case in which it is not clear whether the symptoms are caused by a pure ligamentous sprain or by irritation of the ligaments from carpal subluxation, manipulation may be tried. If it proves ineffective, deep transverse friction is substituted. Even in the chronic stage of a dorsal ligamentous lesion at the wrist, the only effective treatment is deep transverse friction. Other treatments, such as steroid injections, immobilization or operation, are of no use. In order to avoid permanent problems, and because these lesions are often multiple, friction must be given to all the sprained ligaments. Palpation should therefore be performed carefully until all the tender spots have been found.

Technique: deep transverse frictionimage

The patient sits with the hand over the edge of the couch. The therapist sits facing the patient’s wrist, which is flexed with one hand within the limits of pain. Friction is imparted with the other hand. The direction depends on the course of the ligamentous fibres: the radiocarpal and carpometacarpal ligaments are aligned in the same longitudinal axis as the forearm, while the intercarpal ligaments have a more or less transverse direction (Fig. 23.9). For a radiocarpal or carpometacarpal ligament, the direction of the friction is transverse on the ulnar–radial axis. Friction is given with either the index finger, reinforced with the middle finger (Fig. 23.10), or with the thumb. As the lesion is very localized, the therapist must make sure that the finger remains deep between the extensor tendons and does not pass over them.

An intercarpal ligament is given friction with the thumb and in a proximo–distal direction (see Fig. 23.9).

Wrist impaction syndromes

Dorsal and/or ulnar-sided wrist pain may be caused by periosteal bruising that results from impaction between the carpus and the radioulnar border.

Ulnar impaction syndrome

Ulnar impaction syndrome, also known as ulnar abutment or ulnocarpal loading, is a degenerative condition characterized by ulnar wrist pain related to excessive load bearing across the ulnar aspect of the wrist. Chronic impaction between the ulnar head and ulnar carpus results in periostitis and degeneration of the lunate bone, triquetral bone and distal ulnar head, and degenerative tears of the triangular fibrocartilage complex (Fig. 23.11).62

The clinical manifestation of ulnar impaction syndrome generally consists of chronic or subacute ulnar wrist pain, often exacerbated by activity such as loading in extension and relieved by rest. Functional examination shows dorsal pain on passive extension. If the triangular fibrocartilage complex is involved, there will also be pain on forearm rotations.

Radiographic changes include subchondral sclerosis and cystic changes in the ulnar head, ulnar aspect of the proximal lunate bone, and proximal radial aspect of the triquetral bone. MRI is helpful in detecting occult disease in the early stage.63

Treatment will depend on the severity of the lesions. Periostitis responds well to local infiltration with 10 mg of triamcinolone acetonide, provided that further irritation is avoided. This can be done by avoiding the extremes of wrist movement in the loaded position or by the application of a splint or a cast. In more advanced cases or in lesions of the triangular fibrocartilage complex, arthroscopy and surgery will be needed.64,65

Ulnar styloid impaction syndrome

This ulnar-sided wrist pain is caused by impaction between an excessively long ulnar styloid process and the triquetral bone.66 The ulnar styloid process is a continuation of the prominent subcutaneous ridge of the shaft of the ulna, which projects distally towards the triquetral bone for a variable distance (2–6 mm).67 A single or repetitive impaction between the tip of the ulnar styloid process and the triquetral bone results in contusion, which leads to periostitis of the opposing surfaces. If a single-event trauma is forceful enough, fracture of the dorsal triquetral bone may occur.68

The diagnosis of this condition is made on the basis of pinching pain during ulnar deviation and radiographic evidence of an excessively long ulnar styloid process. MRI may show chondromalacia of the ulnar styloid process and proximal triquetral bone (Fig. 23.12).69

Treatment consists of a local infiltration with 10 mg of triamcinolone. In advanced cases, resection of all but the two most proximal millimetres of the styloid process is the treatment of choice.

Radial impaction syndrome

Pain at the dorsal aspect of the wrist may occur as the result of repetitive extension movements during weight bearing, as frequently happens in gymnastics or other high-energy sports; this may lead to periostitis at the distal epiphysis of the radius or the proximal carpal bones,70,71 described as ‘wrist impingement’ syndrome72 or ‘stress reaction’.73 The pain is elicited each time the patient puts weight on an extended wrist.

On examination, passive extension of the wrist is found to be full but painful, all other movements being negative. Careful palpation shows the lesion to lie at the inferior border of the radius or at the scaphoid and lunate bones.

Periostitis responds well to local infiltration with 10 mg of triamcinolone acetonide, provided that further irritation is avoided. This can be done by avoiding the extremes of wrist movement in the loaded position or by the application of a splint or a cast, if necessary. Surgery can be performed in recurrent cases or to avoid long-term complications.74

Repetitive compressive loading may result in stress fractures of the distal part of the radius, the scaphoid and the capitate bones. Scintigraphy and MRI are diagnostic and immobilization in a cast is therapeutic.75,76

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Jun 5, 2016 | Posted by in ORTHOPEDIC | Comments Off on Disorders of the wrist
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