CHAPTER 12 Open treatment of anterior instability—surgical technique
Introduction
With the exception of a high failure rate reported in a series of West Point cadets,3 the results of open stabilization have been uniformly excellent with postoperative recurrence rates generally reported between 0% and 5% (Table 12-1). In this era of evidence-based medicine, three separate meta-analyses have concluded that the results of open stabilization have been superior to those of arthroscopic stabilization.8–10 Of the four direct comparisons of open versus arthroscopic stabilization currently available in the literature,5,11–13 two5,12 of the four revealed clear advantages of the open method. In one13 of the other two reports, no recurrences were found in either group and a large number of patients were excluded based on the findings of an arthroscopic examination, making interpretation of this study difficult. In the final comparison (again from West Point),11 low recurrence rates were noted in both the open and arthroscopic groups, but mean operative time for the open method was 2½ hours compared with less than an hour for the arthroscopic method, suggesting a difference in the authors’ surgical proficiency with the open technique compared with the arthroscopic method. (Our mean operative time for an open stabilization is less than half that.)
N | Recurrence Rate | |
---|---|---|
Gill et al4 | 60 | 5% |
Hubbell et al5 | 20 | 0% |
Pagnani et al6 | 58 | 3% |
Uhorchak et al3 | 66 | 22% |
Wirth et al7 | 142 | 3% |
A concern about the risk of subscapularis rupture after open stabilization has recently been promulgated in two recent reports.14,15 Before the publication of these two series, subscapularis rupture after open stabilization was a reportable case.16,17 These concerns appear to be somewhat overstated. In the series by Sachs et al, only one patient had documented subscapularis insufficiency by magnetic resonance imaging (MRI). In the report by Scheibel et al,15 no complete ruptures were noted on MRI; instead, a degree of atrophy was noted in the superior portion of the tendon that was largely compensated by hypertrophy of the inferior portion. Clinical insufficiency of the subscapularis after open stabilization is exceedingly rare. We have had no instances after primary stabilization in our practice in 15 years. In two recent analyses of shoulder strength after open versus arthroscopic stabilization, strength was equal between the open and arthroscopic groups at 1 year after surgery.18,19
There remain several possible indications for open stabilization, including the following:
We consider two of these indications in the following sections.
Contact athletes
The results of arthroscopic stabilization in contact athletes have been generally disappointing with failures rates ranging from 14% to 60%.5,20–22 Hubbell et al reported no recurrences in contact athletes after open stabilization as compared with a 60% recurrence after arthroscopic stabilization.5
Our experience with open stabilization in 58 American football players yielded a 3% recurrence rate (2 with subluxation, no dislocations).6 Fifty-two of the 58 athletes returned to their sport with only one discontinuing because of the affected shoulder. Motion loss was minimal after open stabilization.
Bony defects of humeral head/glenoid
High recurrence rates have been reported after arthroscopic Bankart repair in patients with bony defects of the glenoid and humeral head.20,23 Burkhart et al20 reported that contact athletes who had an “engaging” Hill-Sachs lesion or “inverted pear” glenoid had a recurrence rate of 67%. Their subdivision of Hill-Sachs lesions into “engaging” and “nonengaging” types is commonly used in contemporary lectures on shoulder instability. Although more recent studies by Sugaya et al24 and Mologne et al25 dispute the contention that high failure rates can be expected after arthroscopic stabilization, it has become accepted in many circles that such lesions increase the risk of failure with arthroscopic capsular repair, and this impression is sometimes generalized to open capsular repair as well.
A review of the few published reports of traditional open capsular repair in the face of defects of the humeral head and/or glenoid reveals that the results have actually been quite good. Rowe et al,17 in their historic 1978 end-result study of open Bankart repairs, found the postoperative recurrence actually decreased, from 3.5% to 2%, in patients with defects of the glenoid rim. Bigliani et al26 reported a 12% recurrence rate after open capsular shift in patients with glenoid bone loss. Rowe et al17 found a slight increase in recurrence after open Bankart repair in patients with moderate or severe Hills-Sachs lesions (5% versus 3.5%). Gill et al,4,27 in a more recent series of open Bankart repairs, found that their recurrence rate doubled from 3% to an acceptable 6% in the presence of a large Hill-Sachs lesion.
We recently determined the recurrence rates with a contemporary method of open anterior stabilization without bony augmentation in patients in our practice with defects of the glenoid and/or humeral head.28 The overall recurrence rate was 2% (2/103), with one patient having a postoperative dislocation and the other experiencing subluxation. The recurrence rate in patients with Hill-Sachs lesions was not statistically higher than the overall rate at 2% (2/87). Patients with engaging Hill-Sachs lesions had a 4% recurrence (1/28), but this was not statistically significant. One of the 9 patients (11%) with large defects of the humeral head had a recurrence—again, not statistically significant. There were no recurrences in the 14 patients with glenoid rim deficiency.
Pathoanatomy of anterior instability
The shoulder has the greatest range of motion of all the joints in the human body. Since bony restraints to motion are minimal, the surrounding soft tissue maintains the humeral head on the glenoid.
The shoulder capsule is large, loose, and redundant. There are three main ligaments in the anterior capsule that help prevent subluxation or dislocation. These ligaments are known as the superior glenohumeral ligament (SGHL), the middle glenohumeral ligament (MGHL), and the inferior glenohumeral ligament complex (IGHLC). Damage to the IGHLC, which supports the inferior part of the shoulder capsule like a hammock, is related to most cases of anterior instability. The Bankart lesion, involving detachment of the IGHLC insertion on the glenoid, is the most common pathologic lesion associated with traumatic anterior instability. Defects or injuries to the SGHL and MGHL also may contribute to instability.29
Preoperative considerations
History
The diagnosis of anterior subluxation is often more subtle. The chief complaint may be a sense of movement, pain, or clicking with certain activities. Pain, rather than instability, may be the predominant complaint. In throwers and other overhead athletes, “dead arm” episodes may occur during which the patient experiences a sharp pain followed by loss of control of the extremity.30
Imaging
Computed tomography (CT) scans may be useful if bony deficiency is noted on plain films. However, the surgeon should be cautioned that CT tends to overestimate the size of larger glenoid lesions and that CT measurement of smaller lesions is not superior to arthroscopic measurement. These phenomena have been noted previously by others.31,32
Indications and contraindications
The indications for surgical treatment of recurrent anterior shoulder instability are highly subjective. They include a desire of the patient to avoid recurrent problems with instability (including the necessity of reporting to the emergency room on a frequent basis to have the shoulder reduced), problems with recurrent pain, an inability to perform certain activities because of a fear of further shoulder instability, and the desire to improve athletic performance with improved shoulder stability. Failure of a thorough trial of nonoperative treatment is also an indication for surgical treatment.
Contraindications to the open technique include voluntary instability and concomitant psychologic disease. Large defects of the humeral head (more than 25% of the articular surface) or of the glenoid (more than 30% of the surface) may require supplemental bone grafting to fill the defects.33 Such lesions are uncommon and, as indicated previously, most defects can be treated with a traditional open repair without bony augmentation.
We prefer to use arthroscopic methods of stabilization in throwing athletes. If an open method is used in this group, we recommend the technique of anterior capsulolabral reconstruction described by Jobe et al34 in which the subscapularis tendon is split rather than detached.