Endoscopic Plantar Fascia Release
Jeremy J. Miles
Michael J. Shereff
ANATOMY
The plantar fascia is composed of sheets of dense collagen bundles located between the skin and superficial muscle layer of the plantar foot. It functions to provide static and dynamic support to the longitudinal arch of the foot. This aponeurosis shares an origin off the anteromedial plantar aspect of the calcaneal tuberosity with the flexor digitorum brevis (deep) and abductor hallucis (medial). Thereafter, the fascia divides into several fibrous slips which insert on the plantar plates of the metatarsophalangeal joints, bases of the proximal phalanges, and the flexor tendon sheaths.
Biomechanically it acts to provide support to the arch of the foot both dynamically and statically. Plantar fascial tension is generated from it being wound around the metatarsal heads to generate a windlass mechanism. Metatarsophalangeal joint dorsiflexion during toe off tensions the plantar fascia, which aids to lock the transverse tarsal joint and provide the stable lever arm needed for efficient gait. Cadaveric studies have shown that transection of the plantar fascia leads to a loss of longitudinal arch height during terminal stance. Plantar fascia transection also alters pressure distribution of the forefoot, thereby increasing the load on the second metatarsal.1, 2
PLANTAR FASCIITIS OVERVIEW
Plantar fasciitis is thought to be the most common cause of plantar heel pain. Other pathologies on the list of differential diagnoses include heel fat pad atrophy, calcaneal stress fractures, and compression neuropathy of the first branch of the lateral plantar nerve. Epidemiologically, men and women are affected equally. Risk factors for developing the disorder include obesity, heel cord tightness, prolonged weight bearing, and repeated trauma on hard surfaces (e.g., road running). The pathogenesis is thought to be repetitive microtears and chronic inflammation of the plantar fascia at its origin.3
Classically, patients give a history of “start-up pain” localized to the medial plantar heel. The pain is worse with the first few steps after getting out of bed or after sitting for a protracted period. At rest, the foot is relaxed in plantar flexion with the metatarsophalangeal joints in slight flexion, resulting in minimal tension on the plantar fascia. When arising from a non-weightbearing position, tension is applied to the plantar fascia leading to microtears and resultant pain. Each time the patient arises after a prolonged time off their feet this process recurs resulting in a chronic nonhealing pathology. Additionally, activity-related plantar fasciitis is often increased with repetitive loading of the heel and toes, especially when performed on hard surfaces.
Physical examination reveals a point of maximal tenderness at the proximal medial origin of the plantar fascia. The trigger point is usually just distal to the area where the soft skin of the medial hindfoot transitions to the thicker, tougher plantar skin on the medial heel. A classic history in conjunction with this finding is generally sufficient to make the clinical diagnosis of plantar fasciitis. Additional workup is rarely necessary for acute plantar fasciitis.
Recalcitrant plantar fasciitis nonresponsive to conservative intervention merits further workup. Routine radiographs are used as a primary investigation of the affected extremity and can be useful to rule out other etiologies of heel pain. Radiographs are also used to assess for predisposing anatomical deformity, or to identify calcification at the origin of the plantar fascia. However, it should be noted that one study looking at radiographs of a random sample of patients encountered a heel spur in 13% of subjects while only one-third of the spurs detected were symptomatic.4 Ultrasound has been shown to be useful in identifying plantar fasciitis when the proximal plantar fascia thickness measures greater than 4 to 5 mm.5, 6 Ultrasound may also be helpful in following treatment as subjective improvement from conservative therapy has been correlated with thinning of the plantar fascia.7 Magnetic resonance imaging (MRI) has also been demonstrated to be useful to measure the plantar fascia thickness, intra- or perifascial edema, fascial tears, and calcaneal edema suggestive of enthesopathy.8, 9 The improved soft tissue detail appreciated with MRI can be used to help rule out more rare soft tissue disorders such as a tumor or abscess.
INDICATIONS
After the clinical diagnosis of plantar fasciitis is made, multimodal conservative treatment is warranted. Generally, first-line treatment consists of plantar fascia, heel cord, and hamstring stretching, which the patient should perform daily.10, 11 Patients are advised to wear soft rubber wedge sole shoes to act as a shock absorbing cushion
and decrease forces to the heel. Soft, flexible inserts with a well to float the heel thereby decrease pressure on the fascial origin and an arch support to diminish the excursion of the plantar fascia during stance phase of gait are recommended. Activities are recommended within the limits of discomfort. Patients are instructed to follow-up at 4- to 6-week intervals for repeat evaluation.
and decrease forces to the heel. Soft, flexible inserts with a well to float the heel thereby decrease pressure on the fascial origin and an arch support to diminish the excursion of the plantar fascia during stance phase of gait are recommended. Activities are recommended within the limits of discomfort. Patients are instructed to follow-up at 4- to 6-week intervals for repeat evaluation.
If a patient returns to clinic with persistent symptoms, the nonsteroidal anti-inflammatory agent may need to be changed. We feel night splints may be helpful, despite their mixed outcomes in randomized studies.12, 13 The use of a plantar fascia strap is another option that some patients may find beneficial.
At the third appointment, custom orthotics designed with soft heel flotation and arch support to decrease plantar fascia stretch while standing may be beneficial.14, 15 These may work better than over-the-counter orthotics as they take into account a patient’s individual anatomy and not simply shoe size. A corticosteroid injection is offered, which can provide relief of varying degrees and duration. Supervised stretching protocols with physical therapy and massage therapy are also prescribed. Physical therapy modalities are of potential benefit and iontophoresis provides another method for localized corticosteroid delivery. Autologous platelet-rich plasma may provide patients with some benefit. However the lack of supporting clinical evidence and the hesitancy of insurance companies to reimburse for the treatment may prohibit general use.
High-energy extracorporeal shock-wave therapy has been shown to improve symptoms in over 80% of patients with chronic plantar fasciitis and can be offered to patients.16, 17 Low-energy or radial shock-wave therapy has also been shown to be effective in recalcitrant cases of plantar fasciitis.18 However, despite good evidence showing their efficacy neither of these treatments are currently reimbursed by insurance. If nonoperative regimens fail to provide relief, the patient is considered to have refractory plantar fasciitis and more invasive measures are indicated.
Plantar fascia release by either open or endoscopic technique is the most commonly utilized treatment for plantar fasciitis refractory to nonsurgical methods. Plantar fascia release has two principle indications. The first and most common indication is chronic recurrent plantar fasciitis pain that has failed conservative measures. The second indication is persistent pain status post traumatic plantar fascia partial tear to complete the release of the medial band of the plantar fascia. Endoscopic plantar fascia release has the benefit of a shorter recovery period and fewer complications compared to the more widely used open release.3, 19, 20
Contraindications to endoscopic plantar fascia release are few. General medical conditions which preclude anesthesia and surgery are the most common contraindication. Prior endoscopic plantar fascia release is also a contraindication as revision surgery, if required, should be performed by open technique. Endoscopic plantar fascia release should not be performed in patients with compromised skin at the portal sites. Also, neurologic or vascular dysfunction of the peripheral distal extremity that may compromise wound healing are contraindications.